#METABOLOMICS WORKBENCH parthosen_20210131_174239 DATATRACK_ID:2440 STUDY_ID:ST001679 ANALYSIS_ID:AN002737 PROJECT_ID:000000
VERSION             	1
CREATED_ON             	February 3, 2021, 9:31 am
#PROJECT
PR:PROJECT_TITLE                 	Quantitative analysis and genome-scale modeling of human CD4+ T-cell
PR:PROJECT_TITLE                 	differentiation reveals subset-specific regulation of glycosphingolipid pathways
PR:PROJECT_TYPE                  	MS: Untargeted and Targeted
PR:PROJECT_SUMMARY               	This project is associated with five different studies(Part 1-5) and each study
PR:PROJECT_SUMMARY               	is associated with one dataset. All the datasets are submitted to Metabolomics
PR:PROJECT_SUMMARY               	Workbench. Part 1/5: It includes untargeted lipidomic analysis of CD4+ T-cell
PR:PROJECT_SUMMARY               	subsets(Th1,Th2,Th17 and iTreg cells) and their paired control(Th0)cells. Part
PR:PROJECT_SUMMARY               	2/5: It includes quantitative targeted measurements of sphingolipids(ceramides
PR:PROJECT_SUMMARY               	and glycosphingolipids) in Th17, iTreg, and their paired control (Th0) cells.
PR:PROJECT_SUMMARY               	Part 3/5: It includes quantitative targeted measurements of
PR:PROJECT_SUMMARY               	sphingolipids(ceramides and glycosphingolipids) in Th17 cells before(scrambled /
PR:PROJECT_SUMMARY               	control) and after the triple knockdown of SPTLC1,2,3 genes (SPT de novo
PR:PROJECT_SUMMARY               	pathway: sphingolipid metabolism). Part 4/5: It includes quantitative targeted
PR:PROJECT_SUMMARY               	measurements of sphingolipids(ceramides, glycosphingolipids) in Th17 cells
PR:PROJECT_SUMMARY               	before (scrambled / control)and after the knockdown of UGCG gene (GCS pathway:
PR:PROJECT_SUMMARY               	sphingolipid metabolism). Part 5/5: It includes measurements of sphingolipids
PR:PROJECT_SUMMARY               	(sphingomyelins) in Th17 cells before (scrambled / control)and after the
PR:PROJECT_SUMMARY               	knockdown of UGCG gene(GCS pathway: sphingolipid metabolism).
PR:INSTITUTE                     	University of Turku
PR:DEPARTMENT                    	Systems Medicine, Turku Bioscience
PR:LABORATORY                    	Metabolomics
PR:LAST_NAME                     	Sen
PR:FIRST_NAME                    	Partho
PR:ADDRESS                       	Tykistökatu 6B, BioCity, 5th Floor, Turku, Southwest, 20521, Finland
PR:EMAIL                         	partho.sen@utu.fi
PR:PHONE                         	0469608145
#STUDY
ST:STUDY_TITLE                   	Quantitative measurements of sphingomyelins in Th17 cells before and after the
ST:STUDY_TITLE                   	knockdown of UGCG gene (GCS pathway: sphingolipid metabolism) (part-V)
ST:STUDY_TYPE                    	MS: Untargeted and targeted analysis
ST:STUDY_SUMMARY                 	Part 5/5: It includes measurements of sphingolipids (sphingomyelins) in Th17
ST:STUDY_SUMMARY                 	cells before (scrambled / control) and after the knockdown of UGCG gene (GCS
ST:STUDY_SUMMARY                 	pathway: sphingolipid metabolism).
ST:INSTITUTE                     	University of Turku
ST:DEPARTMENT                    	Systems Medicine, Turku Bioscience
ST:LABORATORY                    	Metabolomics
ST:LAST_NAME                     	Sen
ST:FIRST_NAME                    	Partho
ST:ADDRESS                       	Tykistökatu 6B, BioCity, 5th Floor, Turku, Southwest, 20521, Finland
ST:EMAIL                         	partho.sen@utu.fi
ST:PHONE                         	0469608145
#SUBJECT
SU:SUBJECT_TYPE                  	Cultured cells
SU:SUBJECT_SPECIES               	Homo sapiens
SU:TAXONOMY_ID                   	9606
SU:GENDER                        	Not applicable
#SUBJECT_SAMPLE_FACTORS:         	SUBJECT(optional)[tab]SAMPLE[tab]FACTORS(NAME:VALUE pairs separated by |)[tab]Raw file names and additional sample data
SUBJECT_SAMPLE_FACTORS           	-	SCr-D1	Treatment:Scrambled | Types:Control	Cell Type=T17; RAW_FILE_NAME=SCr-D1.mzML
SUBJECT_SAMPLE_FACTORS           	-	KD-D1	Treatment:UGCG-knockdown | Types:Test	Cell Type=T17; RAW_FILE_NAME=KD-D1.mzML
SUBJECT_SAMPLE_FACTORS           	-	SCr-D2	Treatment:Scrambled | Types:Control	Cell Type=T17; RAW_FILE_NAME=SCr-D2.mzML
SUBJECT_SAMPLE_FACTORS           	-	KD-D2	Treatment:UGCG-knockdown | Types:Test	Cell Type=T17; RAW_FILE_NAME=KD-D2.mzML
SUBJECT_SAMPLE_FACTORS           	-	SCr-D3	Treatment:Scrambled | Types:Control	Cell Type=T17; RAW_FILE_NAME=SCr-D3.mzML
SUBJECT_SAMPLE_FACTORS           	-	KD-D3	Treatment:UGCG-knockdown | Types:Test	Cell Type=T17; RAW_FILE_NAME=KD-D3.mzML
#COLLECTION
CO:COLLECTION_SUMMARY            	CD4+ T-cells were isolated from human umbilical cord blood as described
CO:COLLECTION_SUMMARY            	previously[1-3]. 1. Ubaid, U. et al. Transcriptional Repressor HIC1 Contributes
CO:COLLECTION_SUMMARY            	to Suppressive Function of Human Induced Regulatory T Cells. Cell Rep 22,
CO:COLLECTION_SUMMARY            	2094-2106, doi:10.1016/j.celrep.2018.01.070 (2018). 2. Khan, M. M. et al. CIP2A
CO:COLLECTION_SUMMARY            	Constrains Th17 Differentiation by Modulating STAT3 Signaling. iScience 23,
CO:COLLECTION_SUMMARY            	100947, doi:10.1016/j.isci.2020.100947 (2020). 3. Tripathi, S. K. et al.
CO:COLLECTION_SUMMARY            	Genome-wide Analysis of STAT3-Mediated Transcription during Early Human Th17
CO:COLLECTION_SUMMARY            	Cell Differentiation. Cell Rep 19, 1888-1901, doi:10.1016/j.celrep.2017.05.013
CO:COLLECTION_SUMMARY            	(2017).
CO:SAMPLE_TYPE                   	T-cells
CO:STORAGE_CONDITIONS            	-80℃
#TREATMENT
TR:TREATMENT_SUMMARY             	For Th17 cell differentiation, isolated CD4+ cells were activated with a
TR:TREATMENT_SUMMARY             	combination of plate-bound anti-CD3 (750 ng/24-well culture plate well;
TR:TREATMENT_SUMMARY             	Immunotech/Beckman Coulter REF # IM-1304) and soluble anti-CD28 ((1ug/mL;
TR:TREATMENT_SUMMARY             	Immunotech/Beckman coulter REF # IM1376) antibodies in serum-free X-Vivo 20
TR:TREATMENT_SUMMARY             	medium (Lonza), in the absence (Th0) or presence (Th17) of IL-6 (20ng/ml, Roche,
TR:TREATMENT_SUMMARY             	Cat# 11138600 001); IL-1β (10ng/ml, R&D Systems Cat # 201 LB); TGF-β1
TR:TREATMENT_SUMMARY             	(10ng/ml, R&D Systems Cat# 240); anti-IL-4 (1 g/ml) R&D Systems Cat# MAB204)
TR:TREATMENT_SUMMARY             	and anti-IFN-γ (1 μg/ml R&D Systems Cat#MAB-285). Differentiation of Th17
TR:TREATMENT_SUMMARY             	cells was confirmed by measuring IL-17 expression by quantitative real-time PCR,
TR:TREATMENT_SUMMARY             	at 72 hours of Th17 / Th0 culturing. For iTreg cell culturing, after of CD25+
TR:TREATMENT_SUMMARY             	cells, done using LD columns and a CD25 depletion kit (Miltenyi Biotec),
TR:TREATMENT_SUMMARY             	CD4+CD25− cells were activated with plate-bound anti-CD3 (500 ng/24-well
TR:TREATMENT_SUMMARY             	culture plate well) and soluble anti-CD28 (500 ng/mL) at a density of 2 × 106
TR:TREATMENT_SUMMARY             	cells/mL of X-vivo 15 serum-free medium (Lonza). For iTreg differentiation, the
TR:TREATMENT_SUMMARY             	medium was supplemented with IL-2 (12 ng/mL), TGF-β (10 ng/mL) (both from R&D
TR:TREATMENT_SUMMARY             	Systems), all-trans retinoic acid (ATRA) (10 nM; Sigma-Aldrich), and human serum
TR:TREATMENT_SUMMARY             	(10%) and cultured at 37°C in 5% CO2. Control Th0 cells were stimulated with
TR:TREATMENT_SUMMARY             	plate-bound anti-CD3 soluble anti-CD28 antibodies without cytokines. For
TR:TREATMENT_SUMMARY             	confirmation of iTreg cell differentiation, we used intracellular staining to
TR:TREATMENT_SUMMARY             	measure, at 72 hours of iTreg culturing, expression of FOXP3 which is the major
TR:TREATMENT_SUMMARY             	transcription factor driving Treg differentiation. Intracellular staining was
TR:TREATMENT_SUMMARY             	performed using buffer sets of Human Regulatory T-cell Staining Kit
TR:TREATMENT_SUMMARY             	(eBioscience/Thermo Fisher Scientific), following the manufacturer’s protocol.
TR:TREATMENT_SUMMARY             	The following antibodies were used: anti-human FOXP3-PE (eBioscience, Cat. No.
TR:TREATMENT_SUMMARY             	12-4776-42) and rat IgG2a isotype control (eBioscience, Cat. No. 72-4321-77A).
TR:TREATMENT_SUMMARY             	All samples were acquired by a flow cytometer (LSRII) and analyzed either with
TR:TREATMENT_SUMMARY             	FlowJo (FLOWJO, LLC) or with Flowing Software. For Th1 and Th2 cells, purified
TR:TREATMENT_SUMMARY             	naive CD4+ T-cells were activated with plate-bound anti-CD3 (500 ng/24-well
TR:TREATMENT_SUMMARY             	culture plate well) and 500 ng/ml soluble anti-CD28 and cultured in the absence
TR:TREATMENT_SUMMARY             	(Th0) or presence of 2.5 ng/ml IL-12 (R&D Systems) (Th1) or 10 ng/ml IL-4 (R&D
TR:TREATMENT_SUMMARY             	Systems) (for Th2). At 48 hours following the activation of the cells, 17 ng/ml
TR:TREATMENT_SUMMARY             	IL-2 (R&D Systems) was added to the cultures. Differentiation of Th1 and Th2
TR:TREATMENT_SUMMARY             	cells was confirmed by measuring (using flow cytometry) the expression of T-bet
TR:TREATMENT_SUMMARY             	and Gata3 at 72 hours after cell activation. Briefly, cells were fixed and
TR:TREATMENT_SUMMARY             	permeabilized using the Intracellular Fixation & Permeabilization Buffer Set
TR:TREATMENT_SUMMARY             	(eBioscience / Thermo Fisher Scientific), according the manufacturer’s
TR:TREATMENT_SUMMARY             	protocol. The following antibodies were used: anti-human GATA3-PE (eBioscience,
TR:TREATMENT_SUMMARY             	12-9966), anti-human T-bet-BV711 (BD, 563320) and corresponding isotype controls
TR:TREATMENT_SUMMARY             	(BV711 Mouse IgG1, BD, 563044 and PE Rat IgG2b, eBioscience, 12-4031-82).
TR:TREATMENT_SUMMARY             	Samples were acquired by BD LSRFortessa™ cell analyzer and data were analyzed
TR:TREATMENT_SUMMARY             	using FlowJo software (FLOWJO, LLC).
#SAMPLEPREP
SP:SAMPLEPREP_SUMMARY            	The frozen cell preps were defrosted on ice. The samples were extracted using a
SP:SAMPLEPREP_SUMMARY            	modified Folch method[1]. 1. Sen, P. et al. Persistent Alterations in Plasma
SP:SAMPLEPREP_SUMMARY            	Lipid Profiles Before Introduction of Gluten in the Diet Associated With
SP:SAMPLEPREP_SUMMARY            	Progression to Celiac Disease. Clin Transl Gastroenterol 10, 1-10,
SP:SAMPLEPREP_SUMMARY            	doi:10.14309/ctg.0000000000000044 (2019). Briefly, 120 µL of cold (4 °C)
SP:SAMPLEPREP_SUMMARY            	extraction solvent (CHCl3: MeOH, (2:1 v/v) was added to the samples. The
SP:SAMPLEPREP_SUMMARY            	extraction solvent containing the following internal standards: C17
SP:SAMPLEPREP_SUMMARY            	Lactosyl(beta) ceramide (D18:1/17:0, 20 ppb), C17 Glucosyl(beta) ceramide
SP:SAMPLEPREP_SUMMARY            	(D18:1/17:0, 20 ppb), C17 ceramide (D18:1/17:0, 20 ppb), C16 ceramide-d7
SP:SAMPLEPREP_SUMMARY            	(d18:1-d7/16:0, 16,57 ppb), C18 ceramide-d7 (d18:1-d7/18:0, 8.75 ppb), C24
SP:SAMPLEPREP_SUMMARY            	ceramide-d7 (d18:1-d7/24:0, 20 ppb), and C24:1 ceramide-d7 (d18:1-d7/24:1(15Z),
SP:SAMPLEPREP_SUMMARY            	9,96 ppb). The samples were the vortexed briefly and left on ice for 30 minutes.
SP:SAMPLEPREP_SUMMARY            	The samples were then centrifuged (9400g, 5 min, 4 °C) and then 60 µL of the
SP:SAMPLEPREP_SUMMARY            	bottom layer was transfer to a clean mass spectrometry vial (2 mL). The samples
SP:SAMPLEPREP_SUMMARY            	were then stored at –80 °C. The samples for this experiments were the same
SP:SAMPLEPREP_SUMMARY            	extracts that the Cer data was acquired from and had SM(18:1/17:0) spiked in
SP:SAMPLEPREP_SUMMARY            	prior to acquisition.
SP:PROCESSING_STORAGE_CONDITIONS 	-80℃
SP:EXTRACT_STORAGE               	-80℃
#CHROMATOGRAPHY
CH:CHROMATOGRAPHY_SUMMARY        	Chromatographic separation was performed on an ACQUITY UHPLC BEH C18 column (2.1
CH:CHROMATOGRAPHY_SUMMARY        	mm × 100 mm, particle size 1.7 µm, Waters, Milford, MA, USA). The flow rate
CH:CHROMATOGRAPHY_SUMMARY        	was set at 0.4 ml/min throughout the run with an injection volume of 1 µL. The
CH:CHROMATOGRAPHY_SUMMARY        	following solvents were used for the gradient elution: Solvent A was H2O with 1%
CH:CHROMATOGRAPHY_SUMMARY        	NH4Ac (1M) and HCOOH (0.1%) added. Solvent B was a mixture of ACN: IPA (1:1 v/v)
CH:CHROMATOGRAPHY_SUMMARY        	with 1% NH4Ac (1M) and HCOOH (0.1%) added. The gradient was programmed as
CH:CHROMATOGRAPHY_SUMMARY        	follows: 0 to 2 min 35-80% B, 2 to 7 min 80-100 % B, 7 to 14 min 100% B. The
CH:CHROMATOGRAPHY_SUMMARY        	column was equilibrated with a 7min period of 35 % B prior to the next run.
CH:CHROMATOGRAPHY_TYPE           	Reversed phase
CH:INSTRUMENT_NAME               	Waters Acquity UHPLC UNSPSC 41115709
CH:COLUMN_NAME                   	Waters BEH C18 (2.1 mm × 100 mm, 1.7 µm )
#ANALYSIS
AN:ANALYSIS_TYPE                 	MS
#MS
MS:INSTRUMENT_NAME               	Bruker impact II UHPLC-QTOF system
MS:INSTRUMENT_TYPE               	QTOF
MS:MS_TYPE                       	ESI
MS:ION_MODE                      	POSITIVE
MS:MS_COMMENTS                   	The SM results for UGCG-silenced Th17 cells data was acquired on a UHPLC-QTOF
MS:MS_COMMENTS                   	system from Bruker (Bruker, Billerica, MA, USA) combining an Elute UHPLC binary
MS:MS_COMMENTS                   	pump and an Impact II system QTOF system. The samples for this experiments were
MS:MS_COMMENTS                   	the same extracts that the Cer data was acquired from and had SM(18:1/17:0)
MS:MS_COMMENTS                   	spiked in prior to acquisition. The data was acquired using the Hystar suite of
MS:MS_COMMENTS                   	software. MZmine 2 was used for all the untargeted data processing.
#MS_METABOLITE_DATA
MS_METABOLITE_DATA:UNITS	ng/ml
MS_METABOLITE_DATA_START
Samples	SCr-D1	KD-D1	SCr-D2	KD-D2	SCr-D3	KD-D3
Factors	Treatment:Scrambled | Types:Control	Treatment:UGCG-knockdown | Types:Test	Treatment:Scrambled | Types:Control	Treatment:UGCG-knockdown | Types:Test	Treatment:Scrambled | Types:Control	Treatment:UGCG-knockdown | Types:Test
SM(40:2)	17.20193205	29.92518117	17.13790764	30.84444737	31.18175593	16.6790407
SM(d39:1)	15.38197985	21.49159303	10.31057802	19.69177511	5.310709225	24.27979387
SM(d42:3)	45.39569106	80.55755523	47.76495437	89.8564094	106.0340446	38.43533259
SM(d34:2)	13.42104561	20.56499258	13.92247586	21.65716417	20.5471264	10.03529777
SM(d18:0/14:0)	4.349973139	6.253309045	4.631394023	7.272072629	6.791737851	3.835228151
SM(d18:1/16:0)	1304.095013	2122.820665	1269.762231	2318.336646	2209.709313	1537.240129
SM(d18:1/24:0)	141.8925289	216.5551407	122.029665	260.1155001	158.6334128	182.1644583
SM(d18:2/24:1)	1.544120503	2.486120427	1.59401539	3.413540223	2.999495106	1.042457274
SM(d32:1)	46.45775613	67.64853456	46.50944152	70.63184664	79.71227573	45.28629093
SM(d33:1)	12.41136815	16.410371	9.863641453	19.59395256	12.384952	17.05606495
SM(d36:1)	127.1885507	187.3470347	111.4172698	216.4558915	146.6283841	144.5802687
SM(d36:2)	7.312437134	11.58058873	6.561771229	8.85432372	7.673800181	7.391628972
SM(d38:2)	2.219475046	3.276988432	2.708171565	3.966326677	5.706922752	2.080261274
SM(d40:1)	147.964426	237.8186666	147.5238876	288.3692518	163.594511	219.4377597
SM(d41:1)	35.13400258	40.66125672	25.71612436	36.95160133	18.61520159	49.42096797
SM(d42:1)	11.55854829	20.9532022	13.78408087	24.36419072	18.10161024	10.42244865
SM(d42:2)	174.692472	311.313033	193.3236548	370.87741	391.5863064	168.6153408
MS_METABOLITE_DATA_END
#METABOLITES
METABOLITES_START
metabolite_name	quantified m/z	RT(minutes)	PubChem ID	KEGG ID
SM(40:2)	785.6485519	6.743978333
SM(d39:1)	773.6486401	7.003965
SM(d42:3)	811.6639938	6.739763333
SM(d34:2)	701.5547943	5.495344167
SM(d18:0/14:0)	677.5545912	5.536829167
SM(d18:1/16:0)	705.5847473	6.077355833
SM(d18:1/24:0)	815.6952006	7.538965833
SM(d18:2/24:1)	811.6638714	6.525891667
SM(d32:1)	675.5390422	5.327025833
SM(d33:1)	689.5547545	5.609240833
SM(d36:1)	731.6017965	6.380199167
SM(d36:2)	729.5850214	5.895625
SM(d38:2)	757.6174533	6.373169167
SM(d40:1)	787.6641947	7.1947175
SM(d41:1)	801.6798416	7.372094167
SM(d42:1)	815.694201	7.2714375
SM(d42:2)	813.6797969	7.122323333
METABOLITES_END
#END