#METABOLOMICS WORKBENCH rjfowle_20241031_114553 DATATRACK_ID:5327 STUDY_ID:ST003570 ANALYSIS_ID:AN005865 PROJECT_ID:PR002203
VERSION             	1
CREATED_ON             	November 12, 2024, 6:18 pm
#PROJECT
PR:PROJECT_TITLE                 	Dietary fructose enhances tumor growth indirectly via interorgan lipid transfer
PR:PROJECT_SUMMARY               	Fructose consumption has increased considerably over the past five decades,
PR:PROJECT_SUMMARY               	largely due to the widespread use of high-fructose corn syrup (HFCS) as a
PR:PROJECT_SUMMARY               	sweetener. It has been proposed that fructose promotes the growth of some tumors
PR:PROJECT_SUMMARY               	by serving as a direct fuel. Here, we show that fructose supplementation
PR:PROJECT_SUMMARY               	enhances tumor growth in animal models of melanoma, breast cancer, and cervical
PR:PROJECT_SUMMARY               	cancer without causing weight gain or insulin resistance. Interestingly, the
PR:PROJECT_SUMMARY               	cancer cells themselves were unable to use fructose readily as a nutrient
PR:PROJECT_SUMMARY               	because they did not express ketohexokinase-C (KHK-C). Primary hepatocytes did
PR:PROJECT_SUMMARY               	express KHK-C, resulting in fructolysis and the excretion of a variety of lipid
PR:PROJECT_SUMMARY               	species, including lysophosphatidylcholines (LPCs). In co-culture experiments,
PR:PROJECT_SUMMARY               	hepatocyte-derived LPCs were consumed by cancer cells and used to generate
PR:PROJECT_SUMMARY               	phosphatidylcholines (PCs), the major phospholipid of cell membranes. In vivo,
PR:PROJECT_SUMMARY               	HFCS supplementation increased several LPC species by >7-fold in serum.
PR:PROJECT_SUMMARY               	Administration of LPCs to mice was sufficient to increase tumor growth.
PR:PROJECT_SUMMARY               	Pharmacological inhibition of ketohexokinase had no direct effect on cancer
PR:PROJECT_SUMMARY               	cells, but it decreased circulating LPC levels and prevented fructose-mediated
PR:PROJECT_SUMMARY               	tumor growth in vivo. These findings reveal that fructose supplementation
PR:PROJECT_SUMMARY               	increases circulating nutrients such as LPCs, which can enhance tumor growth
PR:PROJECT_SUMMARY               	through a cell non-autonomous mechanism.
PR:INSTITUTE                     	Washington University in St. Louis
PR:DEPARTMENT                    	Chemistry
PR:LABORATORY                    	Gary Patti
PR:LAST_NAME                     	Fowle-Grider
PR:FIRST_NAME                    	Ronald
PR:ADDRESS                       	6101 Washington Blvd Unit 202, SAINT LOUIS, MO, 63112, USA
PR:EMAIL                         	rjfowle@wustl.edu
PR:PHONE                         	309-265-7545
#STUDY
ST:STUDY_TITLE                   	Hepatocytes transform fructose into polar metabolites that are selectively
ST:STUDY_TITLE                   	utilized by cancer cells
ST:STUDY_SUMMARY                 	The goal of this project was to determine if hepatocytes can metabolically
ST:STUDY_SUMMARY                 	transform fructose into polar metabolites that CaSki cancer cells can uptake and
ST:STUDY_SUMMARY                 	metabolize. The CaSki cells do not metabolize fructose directly to much extent,
ST:STUDY_SUMMARY                 	but the liver robustly metabolizes fructose. Therefore, we cultured primary
ST:STUDY_SUMMARY                 	hepatocytes from C57BL/6 mice in [U-13C] fructose as the only sugar source for
ST:STUDY_SUMMARY                 	24 hours to form hepatocyte-conditioned media. We then transferred this media to
ST:STUDY_SUMMARY                 	CaSki cells for 24 hours to form CaSki-conditioned media. LC-MS profiling of
ST:STUDY_SUMMARY                 	polar 13C-labeled metabolites of hepatocyte-conditioned media and
ST:STUDY_SUMMARY                 	CaSki-conditioned media revealed that 13C-labeled glucose and 13C-labeled
ST:STUDY_SUMMARY                 	aspartate are released from hepatocytes and are up taken by CaSki cells.
ST:INSTITUTE                     	Washington University in St. Louis
ST:DEPARTMENT                    	Chemistry
ST:LABORATORY                    	Gary Patti
ST:LAST_NAME                     	Fowle-Grider
ST:FIRST_NAME                    	Ronald
ST:ADDRESS                       	5630 Pershing Ave
ST:EMAIL                         	rjfowle@wustl.edu
ST:PHONE                         	3092657545
#SUBJECT
SU:SUBJECT_TYPE                  	Cultured cells
SU:SUBJECT_SPECIES               	Homo sapiens
SU:TAXONOMY_ID                   	9606
#FACTORS
#SUBJECT_SAMPLE_FACTORS:         	SUBJECT(optional)[tab]SAMPLE[tab]FACTORS(NAME:VALUE pairs separated by |)[tab]Raw file names and additional sample data
SUBJECT_SAMPLE_FACTORS           	-	CaSki CM C13 Fructose 1	Sample source:culture media | Conditioned media cell type:CaSki cells	RAW_FILE_NAME(Raw file name)=CaSki CM C13 Fructose 1.mzXML
SUBJECT_SAMPLE_FACTORS           	-	CaSki CM C13 Fructose 2	Sample source:culture media | Conditioned media cell type:CaSki cells	RAW_FILE_NAME(Raw file name)=CaSki CM C13 Fructose 1.mzXML
SUBJECT_SAMPLE_FACTORS           	-	CaSki CM C13 Fructose 3	Sample source:culture media | Conditioned media cell type:CaSki cells	RAW_FILE_NAME(Raw file name)=CaSki CM C13 Fructose 1.mzXML
SUBJECT_SAMPLE_FACTORS           	-	Hepatocyte CM C13 Fructose 1	Sample source:culture media | Conditioned media cell type:Hepatocytes	RAW_FILE_NAME(Raw file name)=Hepatocyte CM C13 Fructose 1.mzXML
SUBJECT_SAMPLE_FACTORS           	-	Hepatocyte CM C13 Fructose 2	Sample source:culture media | Conditioned media cell type:Hepatocytes	RAW_FILE_NAME(Raw file name)=Hepatocyte CM C13 Fructose 2.mzXML
SUBJECT_SAMPLE_FACTORS           	-	Hepatocyte CM C13 Fructose 3	Sample source:culture media | Conditioned media cell type:Hepatocytes	RAW_FILE_NAME(Raw file name)=Hepatocyte CM C13 Fructose 3.mzXML
#COLLECTION
CO:COLLECTION_SUMMARY            	10 mM [U-13C] fructose was dissolved in glucose-free DMEM and 10% dialyzed FBS
CO:COLLECTION_SUMMARY            	with 1% penicillin/streptomycin. The medium was used to culture hepatocytes for
CO:COLLECTION_SUMMARY            	24 hours, yielding hepatocyte-conditioned medium (CM). 50 uL of this
CO:COLLECTION_SUMMARY            	hepatocyte-conditioned medium was then collected and placed in the -80 degree
CO:COLLECTION_SUMMARY            	celsius freezer for subsequent extraction for LC-MS analysis. This
CO:COLLECTION_SUMMARY            	hepatocyte-conditioned medium was then transferred to CaSki cells for 24 hours,
CO:COLLECTION_SUMMARY            	yielding Caski-conditioned medium (CM).50 uL of this CaSki-conditioned medium
CO:COLLECTION_SUMMARY            	was then collected and placed in the -80 degree celsius freezer for subsequent
CO:COLLECTION_SUMMARY            	extraction for LC-MS analysis.
CO:SAMPLE_TYPE                   	Culture Media
#TREATMENT
TR:TREATMENT_SUMMARY             	10 mM [U-13C] fructose was dissolved in glucose-free DMEM and 10% dialyzed FBS
TR:TREATMENT_SUMMARY             	with 1% penicillin/streptomycin. The medium was used to culture hepatocytes for
TR:TREATMENT_SUMMARY             	24 hours, yielding hepatocyte-conditioned medium (CM). This
TR:TREATMENT_SUMMARY             	hepatocyte-conditioned medium was then transferred to CaSki cells for 24 hours,
TR:TREATMENT_SUMMARY             	yielding Caski-conditioned media (CM).
#SAMPLEPREP
SP:SAMPLEPREP_SUMMARY            	90 µL of 2:2:1 methanol:acetonitrile:water (M:A:W) was added to 10 µL of cell
SP:SAMPLEPREP_SUMMARY            	culture media sample. The samples were vortexed and placed at -20 ºC for an
SP:SAMPLEPREP_SUMMARY            	hour. The samples then were centrifuged at 4 ºC for 10 minutes. The supernatant
SP:SAMPLEPREP_SUMMARY            	was removed and analyzed by LC/MS.
#CHROMATOGRAPHY
CH:CHROMATOGRAPHY_SUMMARY        	LC/MS of polar metabolites was performed by using a SeQuant ZIC-pHILIC column
CH:CHROMATOGRAPHY_SUMMARY        	(EMD Millipore) interfaced with an Agilent 6540 Q-TOF. A 150 × 2.1 mm, 5 μm
CH:CHROMATOGRAPHY_SUMMARY        	SeQuant ZIC-pHILIC column was used with an Agilent 1290 Infinity II LC system,
CH:CHROMATOGRAPHY_SUMMARY        	applying methods established previously 65. Mobile-phase solvents had the
CH:CHROMATOGRAPHY_SUMMARY        	following composition: A = 20 mM ammonium acetate in water:acetonitrile (95:5)
CH:CHROMATOGRAPHY_SUMMARY        	and B = 100% acetonitrile. The following linear gradient was used: 0−0.5 min,
CH:CHROMATOGRAPHY_SUMMARY        	90% B; 0.5−30 min, 90−30% B; 30−31 min, 30% B. Injection volumes were 2
CH:CHROMATOGRAPHY_SUMMARY        	μL for all experiments. The column compartment was maintained at 45 °C.
CH:CHROMATOGRAPHY_TYPE           	HILIC
CH:INSTRUMENT_NAME               	Agilent 1290 Infinity II
CH:COLUMN_NAME                   	SeQuant ZIC-pHILIC (150 x 2.1mm,5um)
CH:SOLVENT_A                     	95% water/5% acetonitrile; 20 mM ammonium acetate
CH:SOLVENT_B                     	100% acetonitrile
CH:FLOW_GRADIENT                 	0−0.5 min, 90% B; 0.5−30 min, 90−30% B linear gradient; 30−31 min, 30% B
CH:FLOW_RATE                     	0.2 mL/min
CH:COLUMN_TEMPERATURE            	45
#ANALYSIS
AN:ANALYSIS_TYPE                 	MS
#MS
MS:INSTRUMENT_NAME               	Agilent 6540 QTOF
MS:INSTRUMENT_TYPE               	QTOF
MS:MS_TYPE                       	ESI
MS:ION_MODE                      	NEGATIVE
MS:MS_COMMENTS                   	LC/MS of polar metabolites was performed by using a SeQuant ZIC-pHILIC column
MS:MS_COMMENTS                   	(EMD Millipore) interfaced with an Agilent 6540 Q-TOF. The mass range was set to
MS:MS_COMMENTS                   	50 to 1500 m/z. Instrument parameters were as follows: gas, 200 °C at 4 L/min;
MS:MS_COMMENTS                   	nebulizer, 44 psi at 2000 V; sheath gas, 300 °C at 12 L/min, capillary, 3000 V;
MS:MS_COMMENTS                   	fragmentor, 100 V; skimmer, 65 V; and scan rate, 3 scans/second. The instrument
MS:MS_COMMENTS                   	was operated in negative ionization mode for all samples analyzed. Isotopologues
MS:MS_COMMENTS                   	and 13C enrichment in metabolites of interest were generated from analysis of
MS:MS_COMMENTS                   	the raw data
#MS_METABOLITE_DATA
MS_METABOLITE_DATA:UNITS	Peak area
MS_METABOLITE_DATA_START
Samples	Hepatocyte CM C13 Fructose 1	Hepatocyte CM C13 Fructose 2	Hepatocyte CM C13 Fructose 3	CaSki CM C13 Fructose 1	CaSki CM C13 Fructose 2	CaSki CM C13 Fructose 3
Factors	Sample source:culture media | Conditioned media cell type:Hepatocytes	Sample source:culture media | Conditioned media cell type:Hepatocytes	Sample source:culture media | Conditioned media cell type:Hepatocytes	Sample source:culture media | Conditioned media cell type:CaSki cells	Sample source:culture media | Conditioned media cell type:CaSki cells	Sample source:culture media | Conditioned media cell type:CaSki cells
Fructose	15509061	14057536	14173209	13139699	11743921	11312989
Sorbitol	244091	243313	219755	242023	266051	238526
Glucose	2462097	2915844	2827902	0	0	0
Pyruvate	6893063	7779000	7778540	16450546	21161747	20984056
Lactic Acid	131836628	142800246	144650726	167806324	182577678	216287507
Aspartic Acid	374249	349232	287444	147025	199941	252491
Alanine	93816	96994	96517	1550742	521626	621445
cis-Aconitate	31424	42850	34599	60483	72061	77209
Malic acid	3065301	3276905	3600793	3605861	4221021	5145173
Glutamic Acid	2797055	3104159	1587614	3004677	3309227	4115213
Serine	74186	70015	65568	219277	239625	283614
Alpha-Ketoglutaric Acid	9695092	11054248	11058145	17495973	19086692	22497667
GABA	160742	182988	120544	189621	209198	257238
Succinic acid	103065	114408	111571	281085	336120	430826
Fumaric Acid	229582	245876	269597	271467	332310	430319
Uracil	128525	138755	125994	940351	1125575	1392506
Arginine	39767	41992	50264	34342	43612	46405
Citric acid	3335284	3014980	2841190	2802709	2920624	2601144
MS_METABOLITE_DATA_END
#METABOLITES
METABOLITES_START
metabolite_name	KEGG identifiers
Fructose	C00095
Sorbitol	C00794
Glucose	C00031
Pyruvate	C00022
Lactic Acid	C00186
Aspartic Acid	C00049
Alanine	C00041
cis-Aconitate	C00417
Malic acid	C00149
Glutamic Acid	C00025
Serine	C00065
Alpha-Ketoglutaric Acid	C00026
GABA	C00334
Succinic acid	C00042
Fumaric Acid	C00122
Uracil	C00106
Arginine	C00062
Citric acid	C00158
METABOLITES_END
#END