#METABOLOMICS WORKBENCH weitang_20241026_153558 DATATRACK_ID:5316 STUDY_ID:ST003595 ANALYSIS_ID:AN005903 PROJECT_ID:PR002223
VERSION             	1
CREATED_ON             	November 26, 2024, 5:52 pm
#PROJECT
PR:PROJECT_TITLE                 	Trifluoroacetate reduces plasma lipid levels and the development of
PR:PROJECT_TITLE                 	atherosclerosis in mice
PR:PROJECT_SUMMARY               	Trifluoroacetate (TFA) has been assumed to be an innocuous counterion (to
PR:PROJECT_SUMMARY               	cationic amino acid side chains) present in countless synthetic bioactive
PR:PROJECT_SUMMARY               	peptides and a few FDA-approved therapeutics. We show here that TFA is in fact
PR:PROJECT_SUMMARY               	bioactive and causes dramatic biological effects in multiple strains of mice and
PR:PROJECT_SUMMARY               	cultured human and rat liver cells. In high-fat diet (HFD)-fed low-density
PR:PROJECT_SUMMARY               	lipoprotein receptor-null (LDLr-/-) mice, TFA reduces the levels of plasma
PR:PROJECT_SUMMARY               	cholesterol, triglycerides, and the development of atherosclerotic lesions
PR:PROJECT_SUMMARY               	following either oral or intraperitoneal administration. These physiological
PR:PROJECT_SUMMARY               	effects were observed with TFA alone, or with TFA present as a counterion of a
PR:PROJECT_SUMMARY               	variety of short, unrelated synthetic peptide sequences. Mechanistic
PR:PROJECT_SUMMARY               	investigations including RNA-seq, confocal microscopy, western blotting,
PR:PROJECT_SUMMARY               	metabolomics, proteomics, pharmacokinetics, and biochemical assays indicated
PR:PROJECT_SUMMARY               	that TFA induces peroxisome proliferation by activating peroxisome
PR:PROJECT_SUMMARY               	proliferator-activated receptor (PPAR)-alpha. We confirmed that TFA also caused
PR:PROJECT_SUMMARY               	peroxisome proliferation and downstream phenotypic effects in cultured human and
PR:PROJECT_SUMMARY               	rat liver cells, wild-type C57/Bl mice, and apolipoprotein E-null (apoE-/-)
PR:PROJECT_SUMMARY               	mice, leading to anti-atherosclerotic effects in the latter strain. Given that
PR:PROJECT_SUMMARY               	TFA is a counterion in many peptides employed in early research and development
PR:PROJECT_SUMMARY               	settings, these findings raise the possibility that TFA may be confounding or
PR:PROJECT_SUMMARY               	contributing to phenotypic changes observed in many studies involving peptides.
PR:PROJECT_SUMMARY               	Although our studies suggest that TFA or its analogues might have therapeutic
PR:PROJECT_SUMMARY               	applications, it should be noted that TFA is also a persistent environmental
PR:PROJECT_SUMMARY               	contaminant that is found at high levels in humans relative to other
PR:PROJECT_SUMMARY               	polyfluoroalkyl substances (PFAS), and is a major metabolite following treatment
PR:PROJECT_SUMMARY               	of patients with common inhaled anesthetics, suggesting that the biological
PR:PROJECT_SUMMARY               	effects reported here could have other implications for human health.
PR:INSTITUTE                     	Scripps Research
PR:LAST_NAME                     	Tang
PR:FIRST_NAME                    	Wei
PR:ADDRESS                       	10550 North Torrey Pines Road, La Jolla, California, 92037, USA
PR:EMAIL                         	wtang@scripps.edu
PR:PHONE                         	858-784-2711
#STUDY
ST:STUDY_TITLE                   	Trifluoroacetate reduces plasma lipid levels and the development of
ST:STUDY_TITLE                   	atherosclerosis in mice
ST:STUDY_SUMMARY                 	Trifluoroacetate (TFA) has been assumed to be an innocuous counterion (to
ST:STUDY_SUMMARY                 	cationic amino acid side chains) present in countless synthetic bioactive
ST:STUDY_SUMMARY                 	peptides and a few FDA-approved therapeutics. We show here that TFA is in fact
ST:STUDY_SUMMARY                 	bioactive and causes dramatic biological effects in multiple strains of mice and
ST:STUDY_SUMMARY                 	cultured human and rat liver cells. In high-fat diet (HFD)-fed low-density
ST:STUDY_SUMMARY                 	lipoprotein receptor-null (LDLr-/-) mice, TFA reduces the levels of plasma
ST:STUDY_SUMMARY                 	cholesterol, triglycerides, and the development of atherosclerotic lesions
ST:STUDY_SUMMARY                 	following either oral or intraperitoneal administration. These physiological
ST:STUDY_SUMMARY                 	effects were observed with TFA alone, or with TFA present as a counterion of a
ST:STUDY_SUMMARY                 	variety of short, unrelated synthetic peptide sequences. Mechanistic
ST:STUDY_SUMMARY                 	investigations including RNA-seq, confocal microscopy, western blotting,
ST:STUDY_SUMMARY                 	metabolomics, proteomics, pharmacokinetics, and biochemical assays indicated
ST:STUDY_SUMMARY                 	that TFA induces peroxisome proliferation by activating peroxisome
ST:STUDY_SUMMARY                 	proliferator-activated receptor (PPAR)-alpha. We confirmed that TFA also caused
ST:STUDY_SUMMARY                 	peroxisome proliferation and downstream phenotypic effects in cultured human and
ST:STUDY_SUMMARY                 	rat liver cells, wild-type C57/Bl mice, and apolipoprotein E-null (apoE-/-)
ST:STUDY_SUMMARY                 	mice, leading to anti-atherosclerotic effects in the latter strain. Given that
ST:STUDY_SUMMARY                 	TFA is a counterion in many peptides employed in early research and development
ST:STUDY_SUMMARY                 	settings, these findings raise the possibility that TFA may be confounding or
ST:STUDY_SUMMARY                 	contributing to phenotypic changes observed in many studies involving peptides.
ST:STUDY_SUMMARY                 	Although our studies suggest that TFA or its analogues might have therapeutic
ST:STUDY_SUMMARY                 	applications, it should be noted that TFA is also a persistent environmental
ST:STUDY_SUMMARY                 	contaminant that is found at high levels in humans relative to other
ST:STUDY_SUMMARY                 	polyfluoroalkyl substances (PFAS), and is a major metabolite following treatment
ST:STUDY_SUMMARY                 	of patients with common inhaled anesthetics, suggesting that the biological
ST:STUDY_SUMMARY                 	effects reported here could have other implications for human health.
ST:INSTITUTE                     	Scripps Research
ST:LAST_NAME                     	Tang
ST:FIRST_NAME                    	Wei
ST:ADDRESS                       	10550 North Torrey Pines Road, La Jolla, California, 92037, USA
ST:EMAIL                         	wtang@scripps.edu
ST:PHONE                         	858-784-2711
#SUBJECT
SU:SUBJECT_TYPE                  	Mammal
SU:SUBJECT_SPECIES               	Mus musculus
SU:TAXONOMY_ID                   	10090
#SUBJECT_SAMPLE_FACTORS:         	SUBJECT(optional)[tab]SAMPLE[tab]FACTORS(NAME:VALUE pairs separated by |)[tab]Raw file names and additional sample data
SUBJECT_SAMPLE_FACTORS           	-	PBS1	Sample source:plasma | Treatment:Control	RAW_FILE_NAME(Raw file name)=rp_pos_PBS1_P1-A-4_01_16083.d
SUBJECT_SAMPLE_FACTORS           	-	PBS2	Sample source:plasma | Treatment:Control	RAW_FILE_NAME(Raw file name)=rp_pos_PBS2_P1-A-6_01_16087.d
SUBJECT_SAMPLE_FACTORS           	-	PBS3	Sample source:plasma | Treatment:Control	RAW_FILE_NAME(Raw file name)=rp_pos_PBS3_P1-A-8_01_16091.d
SUBJECT_SAMPLE_FACTORS           	-	PBS4	Sample source:plasma | Treatment:Control	RAW_FILE_NAME(Raw file name)=rp_pos_PBS4_P1-B-1_01_16095.d
SUBJECT_SAMPLE_FACTORS           	-	PBS5	Sample source:plasma | Treatment:Control	RAW_FILE_NAME(Raw file name)=rp_pos_PBS5_P1-B-3_01_16099.d
SUBJECT_SAMPLE_FACTORS           	-	PBS6	Sample source:plasma | Treatment:Control	RAW_FILE_NAME(Raw file name)=rp_pos_PBS6_P1-B-5_01_16105.d
SUBJECT_SAMPLE_FACTORS           	-	PBS7	Sample source:plasma | Treatment:Control	RAW_FILE_NAME(Raw file name)=rp_pos_PBS7_P1-B-7_01_16109.d
SUBJECT_SAMPLE_FACTORS           	-	PBS8	Sample source:plasma | Treatment:Control	RAW_FILE_NAME(Raw file name)=rp_pos_PBS8_P1-B-9_01_16113.d
SUBJECT_SAMPLE_FACTORS           	-	PBS9	Sample source:plasma | Treatment:Control	RAW_FILE_NAME(Raw file name)=rp_pos_PBS9_P1-C-2_01_16117.d
SUBJECT_SAMPLE_FACTORS           	-	PBS10	Sample source:plasma | Treatment:Control	RAW_FILE_NAME(Raw file name)=rp_pos_PBS10_P1-C-4_01_16121.d
SUBJECT_SAMPLE_FACTORS           	-	TFA1	Sample source:plasma | Treatment:TFA	RAW_FILE_NAME(Raw file name)=rp_pos_TFA1_P1-A-3_01_16081.d
SUBJECT_SAMPLE_FACTORS           	-	TFA2	Sample source:plasma | Treatment:TFA	RAW_FILE_NAME(Raw file name)=rp_pos_TFA2_P1-A-5_01_16085.d
SUBJECT_SAMPLE_FACTORS           	-	TFA3	Sample source:plasma | Treatment:TFA	RAW_FILE_NAME(Raw file name)=rp_pos_TFA3_P1-A-7_01_16089.d
SUBJECT_SAMPLE_FACTORS           	-	TFA4	Sample source:plasma | Treatment:TFA	RAW_FILE_NAME(Raw file name)=rp_pos_TFA4_P1-A-9_01_16093.d
SUBJECT_SAMPLE_FACTORS           	-	TFA5	Sample source:plasma | Treatment:TFA	RAW_FILE_NAME(Raw file name)=rp_pos_TFA5_P1-B-2_01_16097.d
SUBJECT_SAMPLE_FACTORS           	-	TFA6	Sample source:plasma | Treatment:TFA	RAW_FILE_NAME(Raw file name)=rp_pos_TFA6_P1-B-4_01_16103.d
SUBJECT_SAMPLE_FACTORS           	-	TFA7	Sample source:plasma | Treatment:TFA	RAW_FILE_NAME(Raw file name)=rp_pos_TFA7_P1-B-6_01_16107.d
SUBJECT_SAMPLE_FACTORS           	-	TFA8	Sample source:plasma | Treatment:TFA	RAW_FILE_NAME(Raw file name)=rp_pos_TFA8_P1-B-8_01_16111.d
SUBJECT_SAMPLE_FACTORS           	-	TFA9	Sample source:plasma | Treatment:TFA	RAW_FILE_NAME(Raw file name)=rp_pos_TFA9_P1-C-1_01_16115.d
SUBJECT_SAMPLE_FACTORS           	-	TFA10	Sample source:plasma | Treatment:TFA	RAW_FILE_NAME(Raw file name)=rp_pos_TFA10_P1-C-3_01_16119.d
#COLLECTION
CO:COLLECTION_SUMMARY            	Blood was collected from female LDLr-/- mice treated by daily oral gavage for
CO:COLLECTION_SUMMARY            	two weeks with a 200 µmol/kg TFA daily dose (n=10) or PBS vehicle (n=10).
CO:COLLECTION_SUMMARY            	Samples were obtained by retro-orbital puncture using a heparinized capillary
CO:COLLECTION_SUMMARY            	tube and then transferred to a K2EDTA collection tube. Plasma was immediately
CO:COLLECTION_SUMMARY            	separated by centrifuging the blood at 5000 rpm for 10 minutes at 4°C.
CO:SAMPLE_TYPE                   	Blood (plasma)
#TREATMENT
TR:TREATMENT_SUMMARY             	Female LDLr-/- mice were treated for two weeks with daily oral gavage of either
TR:TREATMENT_SUMMARY             	200 µmol/kg TFA (n=10) or a PBS vehicle (n=10).
#SAMPLEPREP
SP:SAMPLEPREP_SUMMARY            	Plasma samples (100 µL) from female LDLr-/- mice treated by daily oral gavage
SP:SAMPLEPREP_SUMMARY            	for two weeks with a 200 µmol/kg TFA daily dose (n=10) or PBS vehicle (n=10)
SP:SAMPLEPREP_SUMMARY            	were extracted with cold MeOH (400 µL) and analyzed individually by reversed
SP:SAMPLEPREP_SUMMARY            	phase LC-MS, in both positive and negative mode.
#CHROMATOGRAPHY
CH:CHROMATOGRAPHY_TYPE           	Reversed phase
CH:INSTRUMENT_NAME               	Agilent 1290 Infinity II
CH:COLUMN_NAME                   	Waters ACQUITY UPLC BEH C18 (100 x 1 mm , 1.7 um, 130A)
CH:SOLVENT_A                     	100% water; 0.1% formic acid
CH:SOLVENT_B                     	100% Acetonitrile; 0.1% formic acid
CH:FLOW_GRADIENT                 	0-2min, 1%B; 2-12min, 1%-99%B; 12-15min, 99%B; 15-15.1min, 99%-1%B; 15.1-18min,
CH:FLOW_GRADIENT                 	1%B
CH:FLOW_RATE                     	0.15 mL/min
CH:COLUMN_TEMPERATURE            	25
#ANALYSIS
AN:ANALYSIS_TYPE                 	MS
#MS
MS:INSTRUMENT_NAME               	Bruker Impact II
MS:INSTRUMENT_TYPE               	QTOF
MS:MS_TYPE                       	ESI
MS:ION_MODE                      	POSITIVE
MS:MS_COMMENTS                   	Untargeted metabolomics were performed on an Agilent Technologies 1290 Infinity
MS:MS_COMMENTS                   	II system with an Waters ACQUITY UPLC BEH C18 Column, 130A, 1.7 um, 1 mm X 100
MS:MS_COMMENTS                   	mm, coupled online to a Bruker Impact II QTOF mass spectrometer with
MS:MS_COMMENTS                   	electrospray ionization (ESI) source. The source dry gas temperature was set to
MS:MS_COMMENTS                   	200°C at a flow of 8 L/min. The capillary voltage was set to 4000 V for
MS:MS_COMMENTS                   	positive mode and 5000 V for negative mode and the nebulizer operated at 29 psi.
MS:CAPILLARY_VOLTAGE             	4000 V
MS:DRY_GAS_FLOW                  	8 l/min
MS:DRY_GAS_TEMP                  	200
MS:NEBULIZER                     	29 psi
MS:MS_RESULTS_FILE               	ST003595_AN005903_Results.txt	UNITS:intensity	Has m/z:Yes	Has RT:Yes	RT units:Minutes
#MS_METABOLITE_DATA
MS_METABOLITE_DATA:UNITS	intensity
MS_METABOLITE_DATA_START
Samples	PBS1	PBS2	PBS3	PBS4	PBS5	PBS6	PBS7	PBS8	PBS9	PBS10	TFA1	TFA2	TFA3	TFA4	TFA5	TFA6	TFA7	TFA8	TFA9	TFA10
Factors	Sample source:plasma | Treatment:Control	Sample source:plasma | Treatment:Control	Sample source:plasma | Treatment:Control	Sample source:plasma | Treatment:Control	Sample source:plasma | Treatment:Control	Sample source:plasma | Treatment:Control	Sample source:plasma | Treatment:Control	Sample source:plasma | Treatment:Control	Sample source:plasma | Treatment:Control	Sample source:plasma | Treatment:Control	Sample source:plasma | Treatment:TFA	Sample source:plasma | Treatment:TFA	Sample source:plasma | Treatment:TFA	Sample source:plasma | Treatment:TFA	Sample source:plasma | Treatment:TFA	Sample source:plasma | Treatment:TFA	Sample source:plasma | Treatment:TFA	Sample source:plasma | Treatment:TFA	Sample source:plasma | Treatment:TFA	Sample source:plasma | Treatment:TFA
pantothenic acid	389708.4153	361659.3991	424050.771	312069.7116	359596.9404	519775.3651	394217.2933	285212.0289	385793.8428	407339.936	825258.1191	585664.4188	639193.4057	737358.0059	577863.9958	734607.725	650550.6438	792153.8123	677114.9743	668778.3448
Linoelaidic Acid methyl ester	64455.47029	89251.344	56751.912	59208.8328	51841.1016	62539.14	67892.775	75579.8904	62517.5325	54700.6425	38135.664	25315.92	47396.99571	45758.655	37513.908	48268.46771	30474.90189	31979.0856	26371.7025	66549.0996
Deoxycorticosterone	28215.3595	18357.696	42919.9272	22120.81341	21715.12353	45133.35382	23692.71207	36437.32448	23816.916	26527.98	59516.0653	58937.11513	40874.53535	43182.01628	25546.7844	46236.82758	39042.78404	49246.78696	31026.57157	45827.78945
Dodecanoylcarnitine	44971.6595	97464.79875	80733.43674	57497.63085	52484.42647	104716.552	55534.28667	100445.441	57215.58544	73596.432	41335.42461	45075.39318	56268.6105	73230.49345	38871.9708	39704.04237	33734.33647	49583.09814	31113.50545	42745.84721
LysoPC(15:0)	818958.9359	734167.2326	972045.648	908657.1278	783168.1868	1018081.999	837168.7144	1225117.274	701754.4983	986248.8311	597207.7303	615708.9656	555792.2531	584569.7447	504626.0163	526309.9224	589508.8664	607982.5183	446615.8276	597103.8875
PC(O-16:0/0:0)	1798436.555	1615204.335	1666816.06	1944539.149	1720009.126	1748477.4	1853263.481	1892791.906	1809667.747	1900946.94	1090301.184	985599.72	1013693.075	1036302.229	979680.9154	1012337.003	1078489.579	1035343.808	927285.8832	1248273.974
PC(O-18:1/0:0)	2090044.142	1827920.943	2117310.048	2147154.252	1946043.302	2653339.084	2184222.482	2459802.121	2081835.967	2205287.58	1348154.732	1223932.996	1176459.765	1231377.079	1159142.139	1138870.526	1309319.869	1257407.716	1092967.273	1489429.91
PC(19:1/0:0)	482904.804	390973.3186	631374.912	462023.6797	489758.0756	841191.7952	532674.1216	770747.904	451735.704	580995.7053	370372.8	397704.5268	304205.5236	350052.9969	340658.8344	281876.7343	340454.7062	327362.5212	291519.5598	395260.9843
PC(O-18:0/0:0)	1830748.707	1500021.432	1939281.624	1923133.641	1764514.178	2088900.652	2141415.701	2358607.743	2331328.168	1954778.72	1161335.448	1062634.104	927840.2335	1050907.718	930401.1533	963644.8596	1036530.735	932144.3582	809110.7675	1143340.26
PC(19:0/0:0)	507920.616	456665.8325	655944.965	527930.189	524520.724	731963.9462	598232.7825	756811.857	501263.581	631509.7608	359257.4507	338891.2485	307074.6	317277.813	314195.079	291537.794	318485.7655	307016.3535	244596.8502	374628.1845
PC(20:0/0:0)	2091568.489	1643697.72	2646462.672	1900346.963	2048040.106	2594113.343	2323246.273	2631301.638	2228362.208	2065164.106	1674018.66	1636030.553	1418490.934	1513379.672	1469868.612	1214706.341	1333921.425	1218142.912	1176406.268	1544322.152
PC(O-20)	1216631.547	1253235.312	1293140.52	1253029.044	1111045.55	1434830.019	1684582.768	1653128.665	2023435.885	1225008.344	1009144.961	905214.1932	802051.9945	862615.0255	700368.8321	763830.1117	791264.27	763689.6428	572838.35	1013975.496
LysoPC(22:5)	402767.1657	397718.5443	478298.016	261330.9995	381953.4196	541477.5617	406887.6548	498219.1011	351378.5585	388732.5471	709796.4952	621108.432	549472.2046	610207.0814	566180.1554	670230.0376	592651.273	617526.4811	534108.0622	772666.8441
PC(17:0/0:0)	61572.93825	57821.19235	70096.32	62431.182	63998.704	69723.784	64901.9268	82384.13425	54681.98713	72583.236	45837.288	42179.8248	41179.04375	40930.068	43567.848	40899.936	42027.77775	37202.02602	35924.49648	46526.494
PC(17:0/0:0)2	4146940.963	4155606.467	3822039.749	3489315.933	3335339.594	4006587.903	4518850.648	6544242.988	3184209.189	3946921.069	2058472.083	3107317.657	2727575.507	3032112.247	1956216.285	2092389.663	2122837.994	2907484.186	2380483.354	3324236.435
PC(22:1/0:0)	443034.5233	407509.0663	501315.696	496550.4	451009.3275	575762.974	512361.5625	567078.7875	472783.155	518834.7675	360434.503	331617.0757	303867.648	366556.056	310539.2337	306091.3425	313330.3908	311657.1745	269593.26	378496.1455
PC(33:2)	114355.584	105171.1872	128503.9831	132638.704	118334.5234	125230.2739	100669.5411	161310.54	87499.09387	109964.7731	81765.05847	69328.1053	82679.51238	84226.34123	68892.63566	74474.628	65252.817	69552.44205	66964.14208	74443.7804
MS_METABOLITE_DATA_END
#METABOLITES
METABOLITES_START
metabolite_name		MW	PubChem ID
pantothenic acid		219	6613
Linoelaidic Acid methyl ester		295	5362793
Deoxycorticosterone		346	6166
Dodecanoylcarnitine		344	168381
LysoPC(15:0)		482	24779458
PC(O-16:0/0:0)		482	162126
PC(O-18:1/0:0)			85335863
PC(19:1/0:0)		520	42607444
PC(O-18:0/0:0)		510	2733532
PC(19:0/0:0)		538	24779472
PC(20:0/0:0)		552	24779473
PC(O-20)
LysoPC(22:5)		570	53480475
PC(17:0/0:0)		510	24779463
PC(33:2)		744	52922715
PC(17:0/0:0)2		510	24779463
PC(22:1/0:0)		902	21308787
METABOLITES_END
#END