#METABOLOMICS WORKBENCH oilkay_20241205_115113 DATATRACK_ID:5430 STUDY_ID:ST003647 ANALYSIS_ID:AN005990 PROJECT_ID:PR002257
VERSION                          	1
CREATED_ON                       	01-03-2025
#PROJECT
PR:PROJECT_TITLE                 	Integration of metabolomic and transcriptomic analyses reveals novel regulatory
PR:PROJECT_TITLE                 	functions of the ChREBP transcription factor in energy metabolism.
PR:PROJECT_SUMMARY               	In this study, we have integrated transcriptomic and metabolomic analyses to
PR:PROJECT_SUMMARY               	better understand the role of the metabolic regulatory transcription factor
PR:PROJECT_SUMMARY               	ChREBP in coordinated regulation of key pathways of intermediary metabolism in
PR:PROJECT_SUMMARY               	the liver. We have uncovered regulatory effects of ChREBP on metabolic
PR:PROJECT_SUMMARY               	homeostasis beyond it’s historical role in control of core glucose and lipid
PR:PROJECT_SUMMARY               	metabolic pathways, to now include effects on co-factors, transporters for amino
PR:PROJECT_SUMMARY               	acids and other small molecules, nucleotide metabolism, and control of
PR:PROJECT_SUMMARY               	mitochondrial substrate supply.
PR:INSTITUTE                     	Duke University
PR:LAST_NAME                     	Ilkayeva
PR:FIRST_NAME                    	Olga
PR:ADDRESS                       	300 N Duke St, Durham, NC, 27701, USA
PR:EMAIL                         	olga.ilkayeva@duke.edu
PR:PHONE                         	919-479-2370
PR:DOI                           	http://dx.doi.org/10.21228/M8WC2H
#STUDY
ST:STUDY_TITLE                   	Targeted mass spec-based metabolomic and clinical analyte analyses of liver and
ST:STUDY_TITLE                   	plasma samples from rats with and without hepatic knockdown of ChREBP
ST:STUDY_TITLE                   	expression.
ST:STUDY_SUMMARY                 	The transcription factor Carbohydrate Response Element-Binding Protein (ChREBP)
ST:STUDY_SUMMARY                 	activates genes of glucose, fructose and lipid metabolism in response to
ST:STUDY_SUMMARY                 	carbohydrate feeding. Integrated transcriptomic and metabolomic analyses in rats
ST:STUDY_SUMMARY                 	with GalNac-siRNA-mediated suppression of ChREBP expression (GalNac-siChREBP
ST:STUDY_SUMMARY                 	treatment) in liver revealed novel ChREBP functions relative to rats treated
ST:STUDY_SUMMARY                 	with a GalNac vector expressing a non-targeting siRNA (GalNac-siCtrl treatment).
ST:STUDY_SUMMARY                 	GalNac-siChREBP treatment reduced expression of genes involved in coenzyme A
ST:STUDY_SUMMARY                 	(CoA) biosynthesis, and lowered CoA and short chain acyl CoA levels. Despite
ST:STUDY_SUMMARY                 	suppression of pyruvate kinase, pyruvate levels were maintained, possibly via
ST:STUDY_SUMMARY                 	increased expression of pyruvate and amino acid transporters. In addition,
ST:STUDY_SUMMARY                 	expression of multiple anaplerotic enzymes was decreased by GalNac-siChREBP
ST:STUDY_SUMMARY                 	treatment, affecting TCA cycle intermediates. Finally, GalNAc-siChREBP treatment
ST:STUDY_SUMMARY                 	suppressed late steps in purine and NAD synthesis, with increases in precursors
ST:STUDY_SUMMARY                 	and lowering of end products in both pathways. In sum, our studies have revealed
ST:STUDY_SUMMARY                 	functions of ChREBP beyond its canonical roles in carbohydrate and lipid
ST:STUDY_SUMMARY                 	metabolism to include regulation of substrate transport, mitochondrial function,
ST:STUDY_SUMMARY                 	and cellular energy balance.
ST:INSTITUTE                     	Duke University
ST:LAST_NAME                     	Ilkayeva
ST:FIRST_NAME                    	Olga
ST:ADDRESS                       	300 N Duke St, Durham, NC, 27701, USA
ST:EMAIL                         	olga.ilkayeva@duke.edu
ST:PHONE                         	919-479-2370
ST:SUBMIT_DATE                   	2024-12-05
#SUBJECT
SU:SUBJECT_TYPE                  	Mammal
SU:SUBJECT_SPECIES               	Rattus norvegicus
SU:TAXONOMY_ID                   	10116
#SUBJECT_SAMPLE_FACTORS:         	SUBJECT(optional)[tab]SAMPLE[tab]FACTORS(NAME:VALUE pairs separated by |)[tab]Additional sample data
SUBJECT_SAMPLE_FACTORS           	R01E4-1	1	Treatment:GalNAc-siChrebp | Sample source:liver	RAW_FILE_NAME(AC raw file name)=AnJie_Newgard_AC_013123_a032; RAW_FILE_NAME(AA raw file name)=AnJie_Newgard_AA_013123_a032; RAW_FILE_NAME(OA raw file name)=AnJie_Newgard_27Liver_OA_052924_a020; RAW_FILE_NAME(BCKA raw file name)=AnJie_Newgard_KA_012723_a032; RAW_FILE_NAME(Nucleotides raw file name)=AnJie_Newgard_Liver_NucleoTides_061024_a028; RAW_FILE_NAME(Creatine/Phosphocreatine raw file name)=AnJie_Newgard_27_Liver_061124_a012; RAW_FILE_NAME(SC Acyl CoAs raw file name)=LC_SC_CoA_032123_a018
SUBJECT_SAMPLE_FACTORS           	R01E4-19	19	Treatment:GalNAc-siChrebp | Sample source:liver	RAW_FILE_NAME(AC raw file name)=AnJie_Newgard_AC_013123_a050; RAW_FILE_NAME(AA raw file name)=AnJie_Newgard_AA_013123_a050; RAW_FILE_NAME(OA raw file name)=AnJie_Newgard_27Liver_OA_052924_a038; RAW_FILE_NAME(BCKA raw file name)=AnJie_Newgard_KA_012723_a050; RAW_FILE_NAME(Nucleotides raw file name)=AnJie_Newgard_Liver_NucleoTides_061024_a023; RAW_FILE_NAME(Creatine/Phosphocreatine raw file name)=AnJie_Newgard_27_Liver_061124_a030; RAW_FILE_NAME(SC Acyl CoAs raw file name)=LC_SC_CoA_032123_a036
SUBJECT_SAMPLE_FACTORS           	R01E4-21	21	Treatment:GalNAc-siChrebp | Sample source:liver	RAW_FILE_NAME(AC raw file name)=AnJie_Newgard_AC_013123_a052; RAW_FILE_NAME(AA raw file name)=AnJie_Newgard_AA_013123_a052; RAW_FILE_NAME(OA raw file name)=AnJie_Newgard_27Liver_OA_052924_a040; RAW_FILE_NAME(BCKA raw file name)=AnJie_Newgard_KA_012723_a052; RAW_FILE_NAME(Nucleotides raw file name)=AnJie_Newgard_Liver_NucleoTides_061024_a038; RAW_FILE_NAME(Creatine/Phosphocreatine raw file name)=AnJie_Newgard_27_Liver_061124_a032; RAW_FILE_NAME(SC Acyl CoAs raw file name)=LC_SC_CoA_032123_a038
SUBJECT_SAMPLE_FACTORS           	R01E4-3	3	Treatment:GalNAc-siChrebp | Sample source:liver	RAW_FILE_NAME(AC raw file name)=AnJie_Newgard_AC_013123_a034; RAW_FILE_NAME(AA raw file name)=AnJie_Newgard_AA_013123_a034; RAW_FILE_NAME(OA raw file name)=AnJie_Newgard_27Liver_OA_052924_a022; RAW_FILE_NAME(BCKA raw file name)=AnJie_Newgard_KA_012723_a034; RAW_FILE_NAME(Nucleotides raw file name)=AnJie_Newgard_Liver_NucleoTides_061024_a033; RAW_FILE_NAME(Creatine/Phosphocreatine raw file name)=AnJie_Newgard_27_Liver_061124_a014; RAW_FILE_NAME(SC Acyl CoAs raw file name)=LC_SC_CoA_032123_a020
SUBJECT_SAMPLE_FACTORS           	R01E4-6	6	Treatment:GalNAc-siChrebp | Sample source:liver	RAW_FILE_NAME(AC raw file name)=AnJie_Newgard_AC_013123_a037; RAW_FILE_NAME(AA raw file name)=AnJie_Newgard_AA_013123_a037; RAW_FILE_NAME(OA raw file name)=AnJie_Newgard_27Liver_OA_052924_a025; RAW_FILE_NAME(BCKA raw file name)=AnJie_Newgard_KA_012723_a037; RAW_FILE_NAME(Nucleotides raw file name)=AnJie_Newgard_Liver_NucleoTides_061024_a031; RAW_FILE_NAME(Creatine/Phosphocreatine raw file name)=AnJie_Newgard_27_Liver_061124_a017; RAW_FILE_NAME(SC Acyl CoAs raw file name)=LC_SC_CoA_032123_a023
SUBJECT_SAMPLE_FACTORS           	R01E4-7	7	Treatment:GalNAc-siChrebp | Sample source:liver	RAW_FILE_NAME(AC raw file name)=AnJie_Newgard_AC_013123_a038; RAW_FILE_NAME(AA raw file name)=AnJie_Newgard_AA_013123_a038; RAW_FILE_NAME(OA raw file name)=AnJie_Newgard_27Liver_OA_052924_a026; RAW_FILE_NAME(BCKA raw file name)=AnJie_Newgard_KA_012723_a038; RAW_FILE_NAME(Nucleotides raw file name)=AnJie_Newgard_Liver_NucleoTides_061024_a042; RAW_FILE_NAME(Creatine/Phosphocreatine raw file name)=AnJie_Newgard_27_Liver_061124_a018; RAW_FILE_NAME(SC Acyl CoAs raw file name)=LC_SC_CoA_032123_a024
SUBJECT_SAMPLE_FACTORS           	R01E4-15	15	Treatment:GalNAc-siCtrl | Sample source:liver	RAW_FILE_NAME(AC raw file name)=AnJie_Newgard_AC_013123_a046; RAW_FILE_NAME(AA raw file name)=AnJie_Newgard_AA_013123_a046; RAW_FILE_NAME(OA raw file name)=AnJie_Newgard_27Liver_OA_052924_a034; RAW_FILE_NAME(BCKA raw file name)=AnJie_Newgard_KA_012723_a046; RAW_FILE_NAME(Nucleotides raw file name)=AnJie_Newgard_Liver_NucleoTides_061024_a029; RAW_FILE_NAME(Creatine/Phosphocreatine raw file name)=AnJie_Newgard_27_Liver_061124_a026; RAW_FILE_NAME(SC Acyl CoAs raw file name)=LC_SC_CoA_032123_a032
SUBJECT_SAMPLE_FACTORS           	R01E4-17	17	Treatment:GalNAc-siCtrl | Sample source:liver	RAW_FILE_NAME(AC raw file name)=AnJie_Newgard_AC_013123_a048; RAW_FILE_NAME(AA raw file name)=AnJie_Newgard_AA_013123_a048; RAW_FILE_NAME(OA raw file name)=AnJie_Newgard_27Liver_OA_052924_a036; RAW_FILE_NAME(BCKA raw file name)=AnJie_Newgard_KA_012723_a048; RAW_FILE_NAME(Nucleotides raw file name)=AnJie_Newgard_Liver_NucleoTides_061024_a021; RAW_FILE_NAME(Creatine/Phosphocreatine raw file name)=AnJie_Newgard_27_Liver_061124_a028; RAW_FILE_NAME(SC Acyl CoAs raw file name)=LC_SC_CoA_032123_a034
SUBJECT_SAMPLE_FACTORS           	R01E4-18	18	Treatment:GalNAc-siCtrl | Sample source:liver	RAW_FILE_NAME(AC raw file name)=AnJie_Newgard_AC_013123_a049; RAW_FILE_NAME(AA raw file name)=AnJie_Newgard_AA_013123_a049; RAW_FILE_NAME(OA raw file name)=AnJie_Newgard_27Liver_OA_052924_a037; RAW_FILE_NAME(BCKA raw file name)=AnJie_Newgard_KA_012723_a049; RAW_FILE_NAME(Nucleotides raw file name)=AnJie_Newgard_Liver_NucleoTides_061024_a045; RAW_FILE_NAME(Creatine/Phosphocreatine raw file name)=AnJie_Newgard_27_Liver_061124_a029; RAW_FILE_NAME(SC Acyl CoAs raw file name)=LC_SC_CoA_032123_a035
SUBJECT_SAMPLE_FACTORS           	R01E4-2	2	Treatment:GalNAc-siCtrl | Sample source:liver	RAW_FILE_NAME(AC raw file name)=AnJie_Newgard_AC_013123_a033; RAW_FILE_NAME(AA raw file name)=AnJie_Newgard_AA_013123_a033; RAW_FILE_NAME(OA raw file name)=AnJie_Newgard_27Liver_OA_052924_a021; RAW_FILE_NAME(BCKA raw file name)=AnJie_Newgard_KA_012723_a033; RAW_FILE_NAME(Nucleotides raw file name)=AnJie_Newgard_Liver_NucleoTides_061024_a026; RAW_FILE_NAME(Creatine/Phosphocreatine raw file name)=AnJie_Newgard_27_Liver_061124_a013; RAW_FILE_NAME(SC Acyl CoAs raw file name)=LC_SC_CoA_032123_a019
SUBJECT_SAMPLE_FACTORS           	R01E4-10	10	Treatment:Sal | Sample source:liver	RAW_FILE_NAME(AC raw file name)=AnJie_Newgard_AC_013123_a041; RAW_FILE_NAME(AA raw file name)=-; RAW_FILE_NAME(OA raw file name)=-; RAW_FILE_NAME(BCKA raw file name)=-; RAW_FILE_NAME(Nucleotides raw file name)=-; RAW_FILE_NAME(Creatine/Phosphocreatine raw file name)=-; RAW_FILE_NAME(SC Acyl CoAs raw file name)=-
SUBJECT_SAMPLE_FACTORS           	R01E4-11	11	Treatment:Sal | Sample source:liver	RAW_FILE_NAME(AC raw file name)=AnJie_Newgard_AC_013123_a042; RAW_FILE_NAME(AA raw file name)=-; RAW_FILE_NAME(OA raw file name)=-; RAW_FILE_NAME(BCKA raw file name)=-; RAW_FILE_NAME(Nucleotides raw file name)=-; RAW_FILE_NAME(Creatine/Phosphocreatine raw file name)=-; RAW_FILE_NAME(SC Acyl CoAs raw file name)=-
SUBJECT_SAMPLE_FACTORS           	R01E4-13	13	Treatment:Sal | Sample source:liver	RAW_FILE_NAME(AC raw file name)=AnJie_Newgard_AC_013123_a044; RAW_FILE_NAME(AA raw file name)=-; RAW_FILE_NAME(OA raw file name)=-; RAW_FILE_NAME(BCKA raw file name)=-; RAW_FILE_NAME(Nucleotides raw file name)=-; RAW_FILE_NAME(Creatine/Phosphocreatine raw file name)=-; RAW_FILE_NAME(SC Acyl CoAs raw file name)=-
SUBJECT_SAMPLE_FACTORS           	R01E4-4	4	Treatment:Sal | Sample source:liver	RAW_FILE_NAME(AC raw file name)=AnJie_Newgard_AC_013123_a035; RAW_FILE_NAME(AA raw file name)=-; RAW_FILE_NAME(OA raw file name)=-; RAW_FILE_NAME(BCKA raw file name)=-; RAW_FILE_NAME(Nucleotides raw file name)=-; RAW_FILE_NAME(Creatine/Phosphocreatine raw file name)=-; RAW_FILE_NAME(SC Acyl CoAs raw file name)=-
SUBJECT_SAMPLE_FACTORS           	R01E4-9	9	Treatment:Sal | Sample source:liver	RAW_FILE_NAME(AC raw file name)=AnJie_Newgard_AC_013123_a040; RAW_FILE_NAME(AA raw file name)=-; RAW_FILE_NAME(OA raw file name)=-; RAW_FILE_NAME(BCKA raw file name)=-; RAW_FILE_NAME(Nucleotides raw file name)=-; RAW_FILE_NAME(Creatine/Phosphocreatine raw file name)=-; RAW_FILE_NAME(SC Acyl CoAs raw file name)=-
#COLLECTION
CO:COLLECTION_SUMMARY            	On day 28 between 8 AM-noon, animals were anesthetized and sacrificed for
CO:COLLECTION_SUMMARY            	collection of plasma and tissue samples. To minimize tissue harvest time, a team
CO:COLLECTION_SUMMARY            	of 3 scientists collected tissues from the animals via the following procedure.
CO:COLLECTION_SUMMARY            	Animals were anesthetized with 250 mg/kg Nembutal, and the abdomen and diaphragm
CO:COLLECTION_SUMMARY            	were surgically opened. Five ml of blood was drawn from the heart, and one
CO:COLLECTION_SUMMARY            	operator centrifuged the sample, collected the plasma, and transferred it to
CO:COLLECTION_SUMMARY            	Eppendorf tubes for rapid freezing by submersion in liquid nitrogen. The other
CO:COLLECTION_SUMMARY            	two team members proceeded to immediate surgical excision of the heart and
CO:COLLECTION_SUMMARY            	liver, which were briefly rinsed with ice-cold PBS, quickly wrapped in aluminum
CO:COLLECTION_SUMMARY            	foil, and then frozen by submersion in liquid nitrogen. The time elapsed between
CO:COLLECTION_SUMMARY            	beginning of surgery to removal and freezing of the heart and liver was less
CO:COLLECTION_SUMMARY            	than 2 minutes. Other tissues (skeletal muscle, adipose, kidney) were collected
CO:COLLECTION_SUMMARY            	after excision of the heart and liver and rapidly frozen in liquid nitrogen. All
CO:COLLECTION_SUMMARY            	tissues were stored at -80C until processing for metabolomic, transcriptomic,
CO:COLLECTION_SUMMARY            	and proteomic analyses.
CO:SAMPLE_TYPE                   	Liver
#TREATMENT
TR:TREATMENT_SUMMARY             	All procedures were approved by Duke University Institutional Animal Care and
TR:TREATMENT_SUMMARY             	Use Committee and performed according to the regulations of the committee.
TR:TREATMENT_SUMMARY             	Breeding pairs of Obese Prone CD (OP/CD) Sprague Dawley rats were gifts from Dr.
TR:TREATMENT_SUMMARY             	Warren Grill and Dr. Eric Gonzalez, Duke University, and a colony was
TR:TREATMENT_SUMMARY             	established and maintained by Duke Laboratory Animal Resources (DLAR). Starting
TR:TREATMENT_SUMMARY             	at 4 weeks of age, male OP/CD rats were single-housed with a light cycle of 7 AM
TR:TREATMENT_SUMMARY             	on/7 PM off, and fed ad libitum with a high-fat/high-sucrose (HF/HS) diet
TR:TREATMENT_SUMMARY             	(D12451i, Research Diets) containing 47% fat (kcal) and 17% sucrose (kcal). Body
TR:TREATMENT_SUMMARY             	weight and food intake were monitored weekly. After 9 weeks of feeding of the
TR:TREATMENT_SUMMARY             	HF/HS diet, plasma samples were collected via saphenous vein bleeding. One week
TR:TREATMENT_SUMMARY             	later, animals received an initial subcutaneous injection of one of two
TR:TREATMENT_SUMMARY             	GalNAc-siRNA constructs at a dose of 9 mg/kg body weight, or an equal volume of
TR:TREATMENT_SUMMARY             	the diluent (PBS), (see below for description of the two GalNAc-siRNA reagents).
TR:TREATMENT_SUMMARY             	Additional doses of each GalNAc-siRNA construct were injected at 10, 18 and 25
TR:TREATMENT_SUMMARY             	days after the first injection. Animals were fasted overnight one day after the
TR:TREATMENT_SUMMARY             	third injection (day 19), and subjected to an intraperitoneal glucose tolerance
TR:TREATMENT_SUMMARY             	test (IPGTT) on the following day. Animals were weighed and a glucose solution
TR:TREATMENT_SUMMARY             	(1g/kg body weight) was administered via intraperitoneal injection. Tail blood
TR:TREATMENT_SUMMARY             	samples were obtained and glucose levels measured with a blood glucose meter
TR:TREATMENT_SUMMARY             	(CVSHealth) immediately before and at 30,60, 90, 120, and 180 minutes after
TR:TREATMENT_SUMMARY             	bolus injection of glucose. One day after the fourth GalNAc-siRNA or saline
TR:TREATMENT_SUMMARY             	injection on day 25, plasma samples were collected via saphenous vein bleeding.
TR:TREATMENT_SUMMARY             	A bolus of deuterium oxide (D2O, 10 ml/kg body weight, Sigma Aldrich) was then
TR:TREATMENT_SUMMARY             	given by intraperitoneal injection and followed by free access to drinking water
TR:TREATMENT_SUMMARY             	supplemented with 4% D2O for the rest of the experimental period. Saphenous
TR:TREATMENT_SUMMARY             	plasma samples were collected again one day after the bolus delivery of D2O (day
TR:TREATMENT_SUMMARY             	27).
#SAMPLEPREP
SP:SAMPLEPREP_SUMMARY            	To prepare samples for analyses, frozen livers were pulverized under liquid
SP:SAMPLEPREP_SUMMARY            	nitrogen, and weighed aliquots of the powder (50 mg powdered tissue/aliquot)
SP:SAMPLEPREP_SUMMARY            	were transferred into pre-frozen Eppendorf tubes for further homogenization at
SP:SAMPLEPREP_SUMMARY            	50 mg of wet tissue per 1 ml of homogenate using the following buffers: 50%
SP:SAMPLEPREP_SUMMARY            	acetonitrile/0.3% formic acid for the analysis of amino acids, acylcarnitines,
SP:SAMPLEPREP_SUMMARY            	organic acids, creatine, and phosphocreatine; 3M perchloric acid for the
SP:SAMPLEPREP_SUMMARY            	analysis of branched-chain keto acids; 0.3M perchloric acid for the analysis of
SP:SAMPLEPREP_SUMMARY            	short-chain acyl CoAs; 100% methanol for the analysis of nucleotides.
#CHROMATOGRAPHY
CH:CHROMATOGRAPHY_SUMMARY        	LC-MS/MS analysis of nucleotides
CH:INSTRUMENT_NAME               	Waters Acquity I-Class
CH:COLUMN_NAME                   	EMD Millipore Chromolith FastGradient RP-18e 50-2mm
CH:COLUMN_TEMPERATURE            	40
CH:FLOW_GRADIENT                 	t=0, B=0%; t=22 min., B=40%; t=22.5 min., B=95% followed by 1 min wash and 2 min
CH:FLOW_GRADIENT                 	re-equilibration at the initial conditions.
CH:FLOW_RATE                     	0.3 ml/min
CH:SOLVENT_A                     	95% water, 5% methanol and 5 mM dimethylhexylamine adjusted to pH 7.5 with
CH:SOLVENT_A                     	acetic acid
CH:SOLVENT_B                     	20% water, 80% methanol and 10 mM dimethylhexylamine
CH:CHROMATOGRAPHY_TYPE           	Reversed phase
#ANALYSIS
AN:ANALYSIS_TYPE                 	MS
#MS
MS:INSTRUMENT_NAME               	Waters Xevo TQ-XS
MS:INSTRUMENT_TYPE               	Triple quadrupole
MS:MS_TYPE                       	ESI
MS:MS_COMMENTS                   	All nucleotides were detected in the negative ion MRM mode based on a
MS:MS_COMMENTS                   	characteristic fragmentation reaction as descibed in Gooding JR, Jensen MV, Dai
MS:MS_COMMENTS                   	X, Wenner BR, Lu D, Arumugam R, Ferdaoussi M, MacDonald PE, Newgard CB. Cell
MS:MS_COMMENTS                   	Rep. 2015 Oct 6;13(1):157-167. Adenylosuccinate Is an Insulin Secretagogue
MS:MS_COMMENTS                   	Derived from Glucose-Induced Purine Metabolism. PMID: 26411681. PMCID:
MS:MS_COMMENTS                   	PMC4598307. TargetLynx was used for data processing.
MS:ION_MODE                      	NEGATIVE
#MS_METABOLITE_DATA
MS_METABOLITE_DATA:UNITS         	µM
MS_METABOLITE_DATA_START
Samples	1	19	21	3	6	7	15	17	18	2
Factors	Treatment:GalNAc-siChrebp | Sample source:liver	Treatment:GalNAc-siChrebp | Sample source:liver	Treatment:GalNAc-siChrebp | Sample source:liver	Treatment:GalNAc-siChrebp | Sample source:liver	Treatment:GalNAc-siChrebp | Sample source:liver	Treatment:GalNAc-siChrebp | Sample source:liver	Treatment:GalNAc-siCtrl | Sample source:liver	Treatment:GalNAc-siCtrl | Sample source:liver	Treatment:GalNAc-siCtrl | Sample source:liver	Treatment:GalNAc-siCtrl | Sample source:liver	
Adenylsuccinic acid	1.6009	1.8541	1.3146	1.7545	1.2293	2.3107	5.0981	5.0730	6.5450	3.2187
ADP	44.4280	27.2454	38.4544	44.8900	42.8254	27.8189	44.1720	56.0825	52.8706	39.6590
AICAR	0.0085	0.0167	0.0080	0.0085	0.0223	0.0145	0.0022	0.0031	0.0064	0.0030
AICAR-P	0.1082	0.2847	0.3236	0.1703	0.4690	0.2238	0.0407	0.0215	0.2027	0.0216
AMP	48.6695	39.6468	34.7521	46.6623	34.0174	35.6418	77.3371	88.4368	77.0510	70.2518
ATP	67.2815	25.2783	50.1405	74.5166	77.8041	26.0410	28.0941	46.7540	34.3937	23.9135
GDP	4.3930	2.6993	3.8156	2.8771	3.8972	3.2787	3.6235	5.0288	5.9477	2.7309
GMP	3.0569	3.5743	2.8266	3.4006	2.7854	3.6170	6.6376	6.5458	7.4114	5.9264
GTP	6.7747	2.4657	4.5982	6.2479	6.9886	2.7061	1.9180	3.6526	2.7341	1.9366
IMP	2.4044	1.6775	0.8655	1.6814	1.6302	1.4674	4.3240	4.9645	3.2997	5.9772
NAD	13.7750	10.7725	13.2083	14.9781	13.2631	10.8995	26.8199	30.2760	30.7506	28.8991
NADH	2.2459	2.2419	2.5059	2.4488	1.6473	1.8922	4.0692	5.9545	4.6147	4.3472
NADP	3.1554	1.9423	2.3453	2.5207	2.6916	2.1744	3.6700	4.6046	4.4449	3.9121
NADPH	1.9007	1.4998	1.6460	2.1111	1.8459	1.3165	3.1418	4.8839	3.4649	3.6562
Nicotinic acid mononucleotide	0.0058	0.0054	0.0013	0.0036	0.0020	0.0043	0.0094	0.0178	0.0205	0.0142
Phosphoribosyl pyrophosphate	0.2360	0.1608	0.4017	0.2225	0.4151	0.2519	0.0442	0.1283	0.0749	0.0312
UDP	3.8692	2.9125	4.5861	2.9150	4.6840	3.8725	3.1687	3.1050	4.3978	2.0776
UMP	6.5977	9.1300	6.4680	7.2818	6.8546	7.5459	11.7536	11.5830	14.1381	11.1604
UTP	4.6263	2.0225	4.5624	5.1228	6.6816	2.1882	1.4182	2.0265	1.9487	1.2662
MS_METABOLITE_DATA_END
#METABOLITES
METABOLITES_START
metabolite_name	pubchem_id	inchi_key	kegg_id	other_id	other_id_type	ri	ri_type	moverz_quant	
Adenylsuccinic acid									
ADP									
AICAR									
AICAR-P									
AMP									
ATP									
GDP									
GMP									
GTP									
IMP									
NAD									
NADH									
NADP									
NADPH									
Nicotinic acid mononucleotide									
Phosphoribosyl pyrophosphate									
UDP									
UMP									
UTP									
METABOLITES_END
#END