Summary of project PR000969
This data is available at the NIH Common Fund's National Metabolomics Data Repository (NMDR) website, the Metabolomics Workbench, https://www.metabolomicsworkbench.org, where it has been assigned Project ID PR000969. The data can be accessed directly via it's Project DOI: 10.21228/M8FD5Z This work is supported by NIH grant, U2C- DK119886.
See: https://www.metabolomicsworkbench.org/about/howtocite.php
Project ID: | PR000969 |
Project DOI: | doi: 10.21228/M8FD5Z |
Project Title: | Metabolic and Transcriptomic Signatures of Prenatal Excessive Methionine in Mice |
Project Type: | MS Analysis |
Project Summary: | Micronutrients are key regulators of prenatal one-carbon (C1) metabolism. In this study, we show here that prenatal excessive methionine (MET), a principal micronutrient and methyl-donor, produces in early life stages significant changes in the components of brain C1 pathways and other metabolic pathways including glutamate transmission, mitochondrial function, and lipid metabolism. |
Institute: | University of California, Irvine |
Department: | Department of Pharmaceutical Sciences, School of Medicine; Department of Computer Science, School of Information and Computer Sciences |
Laboratory: | Amal Alachkar Group; Pierre Baldi Group |
Last Name: | Chen |
First Name: | Siwei |
Address: | 66304 Verano Place Rd. South |
Email: | siweic@uci.edu |
Phone: | 2066585923 |
Summary of all studies in project PR000969
Study ID | Study Title | Species | Institute | Analysis(* : Contains Untargted data) | Release Date | Version | Samples | Download(* : Contains raw data) |
---|---|---|---|---|---|---|---|---|
ST001413 | Metabolomic and Transcriptomic Signatures of Prenatal Excessive Methionine in Mice | Mus musculus | University of California, Irvine | MS | 2020-07-24 | 1 | 28 | Uploaded data (2.1G)* |