Summary of project PR000969

This data is available at the NIH Common Fund's National Metabolomics Data Repository (NMDR) website, the Metabolomics Workbench, https://www.metabolomicsworkbench.org, where it has been assigned Project ID PR000969. The data can be accessed directly via it's Project DOI: 10.21228/M8FD5Z This work is supported by NIH grant, U2C- DK119886.

See: https://www.metabolomicsworkbench.org/about/howtocite.php

Project ID: PR000969
Project DOI:doi: 10.21228/M8FD5Z
Project Title:Metabolic and Transcriptomic Signatures of Prenatal Excessive Methionine in Mice
Project Type:MS Analysis
Project Summary:Micronutrients are key regulators of prenatal one-carbon (C1) metabolism. In this study, we show here that prenatal excessive methionine (MET), a principal micronutrient and methyl-donor, produces in early life stages significant changes in the components of brain C1 pathways and other metabolic pathways including glutamate transmission, mitochondrial function, and lipid metabolism.
Institute:University of California, Irvine
Department:Department of Pharmaceutical Sciences, School of Medicine; Department of Computer Science, School of Information and Computer Sciences
Laboratory:Amal Alachkar Group; Pierre Baldi Group
Last Name:Chen
First Name:Siwei
Address:66304 Verano Place Rd. South
Email:siweic@uci.edu
Phone:2066585923

Summary of all studies in project PR000969

Study IDStudy TitleSpeciesInstituteAnalysis
(* : Contains Untargted data)
Release
Date
VersionSamplesDownload
(* : Contains raw data)
ST001413 Metabolomic and Transcriptomic Signatures of Prenatal Excessive Methionine in Mice Mus musculus University of California, Irvine MS 2020-07-24 1 28 Uploaded data (2.1G)*
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