Summary of project PR001668

This data is available at the NIH Common Fund's National Metabolomics Data Repository (NMDR) website, the Metabolomics Workbench, https://www.metabolomicsworkbench.org, where it has been assigned Project ID PR001668. The data can be accessed directly via it's Project DOI: 10.21228/M83H7N This work is supported by NIH grant, U2C- DK119886.

See: https://www.metabolomicsworkbench.org/about/howtocite.php

Project ID: PR001668
Project DOI:doi: 10.21228/M83H7N
Project Title:ATP10A promotes endothelial cell insulin sensitivity and protects against female-specific dyslipidemia
Project Type:MS Untargeted Lipidomics
Project Summary:The role of ATP10A in endothelial dysfunction
Institute:Vanderbilt University
Department:Biological Sciences
Laboratory:Graham
Last Name:Graham
First Name:Todd
Address:5260 Medical Research Building III BSB
Email:tr.graham@Vanderbilt.Edu
Phone:615-343-1835
Publications:Norris, A.C., Yazlovitskaya, E., Zhu, L., Rose, B., Yu, S., McLean, J.A., Stafford, J. and Graham, T.R., 2022. ATP10A has a protective and sex‚Äźspecific role in lipid metabolism. The FASEB Journal, 36.

Summary of all studies in project PR001668

Study IDStudy TitleSpeciesInstituteAnalysis
(* : Contains Untargted data)
Release
Date
VersionSamplesDownload
(* : Contains raw data)
ST002696 ATP10A promotes endothelial cell insulin sensitivity and protects against female-specific dyslipidemia Mus musculus Vanderbilt University MS* 2023-05-24 1 40 Uploaded data (90.3G)*
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