Studies involving disease:Sepsis

Study ID Study Title Species Institute
ST000785Pharmacometabolomics of L-Carnitine Treatment Response Phenotypes in Patients with Septic ShockHumanUniversity of Michigan
ST001101Identification of potential sepsis biomarkers in burn injury conditions.MouseTexas Tech University
ST001190Sepsis-related metabolic changes in ileum, jejunum, skeletal muscle, liver and lungPigIndiana University School of Medicine
ST001255Immunomodulatory activity of hyaluronidase is associated with metabolic adaptations during acute inflammationMouseSao Paulo University
ST001294Estimating Platelet Mitochondrial Function in Patients with Sepsis - Platelet NMRs (part-I)HumanUniversity of Michigan; University of Mississippi; University of Minnesota
ST001295Estimating Platelet Mitochondrial Function in Patients with Sepsis - WB NMRs (part-II)HumanUniversity of Michigan; University of Mississippi; University of Minnesota
ST001319Pre-treatment L-Carnitine Pharmacometabolomics in Sepsis (CaPS) PatientsHumanUniversity of Michigan
ST00135448 hours post-treatment L-Carnitine Pharmacometabolomics in Sepsis (CaPS) PatientsHumanUniversity of Michigan
ST001390Natural history of the systemic responses to a renal inoculation of uropathogenic E. coli in swinePigUniversity of Michigan
ST001499Metabolomics reveals the protective effect of isosteviol sodium against multiple organ injury in septic mice - HeartMouseGuangdong University of Technology
ST001500Metabolomics reveals the protective effect of isosteviol sodium against multiple organ injury in septic mice - LiverMouseGuangdong University of Technology
ST001502Metabolomics reveals the protective effect of isosteviol sodium against multiple organ injury in septic mice - LungMouseGuangdong University of Technology
ST001503Metabolomics reveals the protective effect of isosteviol sodium against multiple organ injury in septic mice - KidneyMouseGuangdong University of Technology
ST001504Metabolomics reveals the protective effect of isosteviol sodium against multiple organ injury in septic mice - SpleenMouseGuangdong University of Technology
ST001505Metabolomics reveals the protective effect of isosteviol sodium against multiple organ injury in septic mice - PlasmaMouseGuangdong University of Technology
ST002455Organism-Wide Analysis of Sepsis Reveals Mechanisms of Systemic InflammationMouseUniversity of Chicago
ST003136Circulating N-lactoyl-amino acids and N-formyl-methionine reflect mitochondrial dysfunction and predict mortality in septic shockHumanMassachusetts General Hospital
ST003189Untargeted Metabolomic Profile of Septic Shock in the Emergency DepartmentHumanNational Taiwan University
ST003439Metabolomics of human serum to support understanding of early metabolic shift in presymptomatic sepsis patients. (Part1 human metabolites)HumanLeibniz Institute for Natural Product Research and Infection Biology Hans Knöll Institute
ST003456Lipidomics of human serum to support understanding of early metabolic shift in presymptomatic sepsis patients. (Part2 human lipids)HumanLeibniz Institute for Natural Product Research and Infection Biology Hans Knöll Institute
ST003457Metabolomics of mice heart to support understanding of early metabolic shift in presymptomatic sepsis patients. (Part3 mice heart metabolite)MouseLeibniz Institute for Natural Product Research and Infection Biology Hans Knöll Institute
ST003458Metabolomics of mice kidney to support understanding of early metabolic shift in presymptomatic sepsis patients. (Part4 mice kidney metabolite)MouseLeibniz Institute for Natural Product Research and Infection Biology Hans Knöll Institute
ST003459Metabolomics of mice liver to support understanding of early metabolic shift in presymptomatic sepsis patients. (Part5 mice liver metabolite)MouseLeibniz Institute for Natural Product Research and Infection Biology Hans Knöll Institute
ST003460Metabolomics of mice serum to support understanding of early metabolic shift in presymptomatic sepsis patients. (Part6 mice serum metabolite)MouseLeibniz Institute for Natural Product Research and Infection Biology Hans Knöll Institute
ST003461Metabolomics of mice spleen to support understanding of early metabolic shift in presymptomatic sepsis patients. (Part7 mice spleen metabolite)MouseLeibniz Institute for Natural Product Research and Infection Biology Hans Knöll Institute
ST003462Metabolomics of mice white adipose tissue to support understanding of early metabolic shift in presymptomatic sepsis patients. (Part8 mice white adipose tissue metabolite)MouseLeibniz Institute for Natural Product Research and Infection Biology Hans Knöll Institute
ST003464Lipidomics of mice heart to support understanding of early metabolic shift in presymptomatic sepsis patients. (Part9 mice heart lipid)MouseLeibniz Institute for Natural Product Research and Infection Biology Hans Knöll Institute
ST003465Lipidomics of mice kidney to support understanding of early metabolic shift in presymptomatic sepsis patients. (Part10 mice kidney lipid)MouseLeibniz Institute for Natural Product Research and Infection Biology Hans Knöll Institute
ST003466Lipidomics of mice liver to support understanding of early metabolic shift in presymptomatic sepsis patients. (Part11 mice liver lipid)MouseLeibniz Institute for Natural Product Research and Infection Biology Hans Knöll Institute
ST003467Lipidomics of mice serum to support understanding of early metabolic shift in presymptomatic sepsis patients. (Part12 mice serum lipid)MouseLeibniz Institute for Natural Product Research and Infection Biology Hans Knöll Institute
ST003468Lipidomics of mice spleen to support understanding of early metabolic shift in presymptomatic sepsis patients. (Part13 mice spleen lipid)MouseLeibniz Institute for Natural Product Research and Infection Biology Hans Knöll Institute
ST003469Lipidomics of mice white adipose tissue to support understanding of early metabolic shift in presymptomatic sepsis patients. (Part14 mice white adipose tissue lipid)MouseLeibniz Institute for Natural Product Research and Infection Biology Hans Knöll Institute
ST003980Glutamate-mediated TCA anaplerosis is increased in septic mitochondriaMouseGhent University
ST003981Mitochondrial pyruvate metabolism is disturbed in sepsisMouseGhent University
ST003982Mitochondrial pyruvate metabolism is disturbed and contributes to hyperlactatemia and lethality in sepsis - Liver, Plasma, Skeletal muscleMouseGhent University
ST003983Mitochondrial pyruvate metabolism is disturbed and contributes to hyperlactatemia and lethality in sepsis - Lung, Heart, Spleen, Kidney, RBCMouseGhent University

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