Summary of Study ST001894
This data is available at the NIH Common Fund's National Metabolomics Data Repository (NMDR) website, the Metabolomics Workbench, https://www.metabolomicsworkbench.org, where it has been assigned Project ID PR001192. The data can be accessed directly via it's Project DOI: 10.21228/M8MX3X This work is supported by NIH grant, U2C- DK119886.
See: https://www.metabolomicsworkbench.org/about/howtocite.php
| Study ID | ST001894 |
| Study Title | Involvement of Mieap in cardiolipin metabolism (part II) |
| Study Summary | Mass spectrometric data of Cardiolipin in Mice kidney (Mieap-WT vs. Mieap-KO), and Mice liver (Mieap-WT vs. Mieap-KO) |
| Institute | National Cancer Center Japan Research Institute |
| Last Name | Ikari |
| First Name | Naoki |
| Address | 5-1-1 Tsukiji, Chuo-ku, Tokyo 104-0045, Japan |
| nikari@ncc.go.jp | |
| Phone | +81-3-3542-2511 |
| Submit Date | 2021-07-30 |
| Analysis Type Detail | LC-MS |
| Release Date | 2021-08-09 |
| Release Version | 1 |
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Treatment:
| Treatment ID: | TR001984 |
| Treatment Summary: | The Mieap-knockout (Mieap-/-) mice were generated by using the Cre/loxP recombination system as previously reported[1]. Briefly, the floxed and trapped alleles were generated using a single construct bearing a gene-trap cassette doubly flanked by LoxP and FRT located between exons 5 and 8 of the mouse Mieap gene, which is located on chromosome 5. The Mieap homozygous (Mieap-/-) deficient mice were generated by mating breeding pairs of the Mieap heterozygous (Mieap+/-) mice. All mice were housed at 22 ± 2°C with a 12 h light/dark cycle with free access to food, CE-2 (CLEA Japan) and water. [1]Tsuneki, M., Nakamura, Y., Kinjo, T., Nakanishi, R., and Arakawa, H. (2015). Mieap suppresses murine intestinal tumor via its mitochondrial quality control. Sci. Rep. 5, 12472. |
| Treatment: | Knockout |
| Treatment Compound: | None |
| Animal Fasting: | None |
